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The immunomodulatory effect of laquinimod in CNS autoimmunity is mediated by the aryl hydrocarbon receptor.
Aquaporin modulators: a patent review (2010-2015).
Vertebral hemangiomas in the thoracic spine of multiple sclerosis patients are connected with fewer demyelinating lesions at the same level. Possible impact on pathophysiology and clinical course.
Striving for Balance Between Caring and Restraint: Young Adults' Experiences with Parental Multiple Sclerosis.
To fingolimod and beyond: The rich pipeline of drug candidates that target S1P signaling.
Efficacy and Safety of Rituximab Therapy in Neuromyelitis Optica Spectrum Disorders: A Systematic Review and Meta-analysis.
Physical comorbidities increase the risk of psychiatric comorbidity in multiple sclerosis.
The increased antibody response to Epstein-Barr virus in multiple sclerosis is restricted to selected virus proteins.
Multiple Sclerosis (MS) in the Life Cycle of the Family: An Interpretative Phenomenological Analysis of the Perspective of Persons With Recently Diagnosed MS.
The impact of adjusted work conditions and disease-modifying drugs on work ability in multiple sclerosis.
Blockade of glutamine synthetase enhances inflammatory response in microglial cells.
Partial MHC class II constructs as novel immunomodulatory therapy for stroke.
Natalizumab treatment leads to an increase in circulating CXCR3-expressing B cells.
Improving the management of chronic inflammatory demyelinating polyradiculoneuropathy.
MicroRNAs associated with the pathogenesis of multiple sclerosis.
Acute inflammatory demyelinating polyradiculoneuropathy in a newborn infant.
Enterovirus infections in Singaporean children: an assessment of neurological manifestations and clinical outcomes.
Multimodal evoked potentials for functional quantification and prognosis in multiple sclerosis.
Stent-assisted coil embolization of challenging intracranial aneurysms: initial and mid-term results with low-profile ACCLINO devices.
Loss of ceramide synthase 2 activity, necessary for myelin biosynthesis, precedes tau pathology in the cortical pathogenesis of Alzheimer's disease.
Why use sex hormones in relapsing-remitting multiple sclerosis?
Healthcare Costs and Resource Utilization in Patients with Multiple Sclerosis Relapses Treated with H.P. Acthar Gel(®).
Complement is activated in progressive multiple sclerosis cortical grey matter lesions.
Glutamate signalling: A multifaceted modulator of oligodendrocyte lineage cells in health and disease.
Dietary salt intake and time to relapse in paediatric multiple sclerosis.
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