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Most Viewed
Progressive MS: from pathophysiology to drug discovery.
Inflammation Modulates RLIP76/RALBP1 Electrophile-Glutathione Conjugate Transporter and Housekeeping Genes in Human Blood-Brain Barrier Endothelial Cells.
Development of a neuroprotective peptide that preserves survival pathways by preventing Kidins220/ARMS calpain processing induced by excitotoxicity.
Using conceptual work products of case management to design health IT.
Optic neuritis as an initial manifestation of human herpesvirus 6 reactivation after unrelated bone marrow transplantation.
Human neural progenitor cells in central nervous system lesions.
Transitions between SNF and home-based care in patients with multiple sclerosis.
Olanzapine ameliorates neuropathological changes and increases IGF-1 expression in frontal cortex of C57BL/6 mice exposed to cuprizone.
Mining gene expression data of multiple sclerosis.
JC polyomavirus attachment, entry, and trafficking: unlocking the keys to a fatal infection.
Estimation of the multiple sclerosis risk in population living on contaminated territories after the Chornobyl catastrophe.
First-dose effects of fingolimod: Pooled safety data from three phase 3 studies.
Early diagnosis of Balo's concentric sclerosis by diffusion tensor tractography: a case report and literature review.
Acid-sensing ion channels promote the inflammation and migration of cultured rat microglia.
Immune Responses to Non-Tumor Antigens in the Central Nervous System.
Seeking balance: Potentiation and inhibition of multiple sclerosis autoimmune responses by IL-6 and IL-10.
Assessment of Optic Nerve Impairment in Patients with Neuromyelitis Optica by MR Diffusion Tensor Imaging.
Quality of life in patients with multiple sclerosis in Republic of Srpska.
Clinical prospective study of visual function in patients with acute optic neuritis.
Upregulation of ciliary neurotrophic factor in astrocytes by aspirin: Implications for remyelination in multiple sclerosis.
Fingolimod therapy for multiple sclerosis.
[Diagnosis and differential diagnosis of demyelinating optic neuropathy in multiple sclerosis.]
Loss of branched o-mannosyl glycans in astrocytes accelerates remyelination.
Cytometric profiling in various clinical forms of multiple sclerosis with respect to CD21+, CD32+, and CD35+ B and T cells.
Loss of astrocytic leptin signaling worsens experimental autoimmune encephalomyelitis.
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