Multiple Sclerosis Discovery Forum

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ALCAM and CD6 - multiple sclerosis risk factors.
The treatment of neuromyelitis optica.
Satellite glial cells in dorsal root ganglia are activated in experimental autoimmune encephalomyelitis.
Coenzyme Q10 supplementation and multiple sclerosis.
A clinical and radiological profile of neuromyelitis optica and spectrum disorders in an Indian cohort.
Quantitative analysis of the suppressors of cytokine signaling 1 and 3 in peripheral blood leukocytes of patients with multiple sclerosis.
Peroxisome proliferator-activated receptor-γ cofactors in neurodegeneration.
[Assessment of the high cost drug program on an example of the interferon treatment of multiple sclerosis.]
The amyloid state of proteins in human diseases.
Magnitude and concurrence of anxiety and depression among attendees with multiple sclerosis at a tertiary care Hospital in Oman.
Quality of life and cognitive functions in early onset multiple sclerosis.
Immunophenotype of mouse cerebral hemispheres-derived neural precursor cells.
Erratum.
Programmed Death Ligand-1 on Microglia Regulates Th1 Differentiation via Nitric Oxide in Experimental Autoimmune Encephalomyelitis.
Metabolic programming and PDHK1 control CD4+ T cell subsets and inflammation.
Detection of JCPyV microRNA in blood and urine samples of multiple sclerosis patients under natalizumab therapy.
Mind the gap! Lay and medical perceptions of risks associated with the use of alternative treatment and conventional medicine.
Adrenomedullin protects from experimental autoimmune encephalomyelitis at multiple levels.
T cell-activation in neuromyelitis optica lesions plays a role in their formation.
Multiple Sclerosis and Antibodies against KIR4.1.
Botulinum Toxin Treatment of Spasticity in Adults and Children.
Dual roles of the adenosine A2a receptor in autoimmune neuroinflammation.
Vitamin D Status During Pregnancy and Risk of Multiple Sclerosis in Offspring of Women in the Finnish Maternity Cohort.
Switch-Associated Protein 70 Antibodies in Multiple Sclerosis: Possible Association with Disease Progression.
Selective accumulation of pro-inflammatory T cells in the intestine contributes to the resistance to autoimmune demyelinating disease.

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