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The majority of CD1d-sulfatide-specific T cells in human blood use a semiinvariant Vδ1 TCR.
IL-10 signaling is essential for 1,25-dihydroxyvitamin D3-mediated inhibition of experimental autoimmune encephalomyelitis.
Treating progressive multifocal leukoencephalopathy with interleukin 7 and vaccination with JC virus capsid protein VP1.
Pharmacogenetic study of long-term response to interferon-β treatment in multiple sclerosis.
Brain reserve and cognitive reserve in multiple sclerosis: What you've got and how you use it.
Pathological mechanisms in progressive multiple sclerosis.
The topograpy of demyelination and neurodegeneration in the multiple sclerosis brain.
Daclizumab reduces CD25 levels on T cells through monocyte-mediated trogocytosis.
Good Early Results for HERV Suppressor in MS
Microglia activation is associated with IFN-α induced depressive-like behavior.
In vivo gene expression revealed by cDNA arrays: the pattern in relapsing-remitting multiple sclerosis patients compared with normal subjects.
Assessment of Disease Activity in Multiple Sclerosis Phenotypes with Combined Gadolinium- and Superparamagnetic Iron Oxide-enhanced MR Imaging.
Title: Pharmacokinetics and Pharmacodynamics of Peginterferon Beta-1a in Patients with Relapsing-Remitting Multiple Sclerosis in the Randomised ADVANCE Study.
First proof for rituximab in IgG4-related disease
Campath-1H treatment of multiple sclerosis: lessons from the bedside for the bench.
Mountain View Pharmaceuticals receives European patent on potential long-acting drug for multiple sclerosis
1,25-dihydroxyvitamin D(3) ameliorates Th17 autoimmunity via transcriptional modulation of interleukin-17A.
Restoration of axon conduction and motor deficits by therapeutic treatment with glatiramer acetate.
Colestipol May Quickly Eliminate Teriflunomide
Immunology 2014
Company restores access to multiple sclerosis drug after pressure from neurologists.
Intellectual enrichment lessens the effect of brain atrophy on learning and memory in multiple sclerosis.
Cladribine
Regional Susceptibility to TNF-α Induction of Murine Brain Inflammation via Classical IKK/NF-κB Signalling.
Immunomodulatory drugs (natalizumab), worsening of multiple sclerosis, rebound effect and similitude.
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