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Most Viewed Papers
Endogenous and recombinant type I interferons and disease activity in multiple sclerosis.
Reversible cerebral vasoconstriction syndrome associated with fingolimod treatment in relapsing-remitting multiple sclerosis three months after childbirth.
Regulation of encephalitogenicity of neuroantigen-primed T cells by nitric oxide: Implications for multiple sclerosis.
Multiple sclerosis shrinks intralesional, and enlarges extralesional, brain parenchymal veins.
Multi-compartment t2 relaxometry using a spatially constrained multi-gaussian model.
Elevated melatonin levels in natalizumab-treated female patients with relapsing-remitting multiple sclerosis: Relationship to oxidative stress.
[HTLV-1-associated myelopathy/tropical spastic paraparesis : A differential diagnosis in multiple sclerosis.]
Riluzole inhibits spontaneous Ca2+ signaling in neuroendocrine cells by activation of K+ channels and inhibition of Na+ channels.
Assessment of changes in immune measures of multiple sclerosis patients treated with laquinimod.
CD8+ T-Cell Deficiency, Epstein-Barr Virus Infection, Vitamin D Deficiency, and Steps to Autoimmunity: A Unifying Hypothesis.
Mast Cell-Mediated Inhibition of Abdominal Neutrophil Inflammation by a PEGylated TLR7 Ligand.
Unraveling Natalizumab Effects on Deregulated miR-17 Expression in CD4(+) T Cells of Patients with Relapsing-Remitting Multiple Sclerosis.
Multiple sclerosis.
Using experimental autoimmune encephalomyelitis as a model to study the effect of prenatal stress on fetal programming.
Astrocyte proliferation is regulated by the OGF-OGFr axis in vitro and in experimental autoimmune encephalomyelitis.
Very early MS -- insights from MRI.
Efficacy and safety of Alemtuzumab in treatment-naive patients with relapsing-remitting multiple sclerosis: Four-year follow-up of the Care-MS I study.
Potential role of OPG/RANKL system and FokI genotypes in pathogenesis and clinical manifestations in multiple sclerosis.
In vivo vs. ex vivo Magnetic Resonance Imaging In Mice.
Natalizumab treatment reduces endothelial activity in MS patients.
Multiple sclerosis relapses and depression.
TRPM4 cation channel mediates axonal and neuronal degeneration in experimental autoimmune encephalomyelitis and multiple sclerosis.
Immune surveillance in multiple sclerosis patients treated with natalizumab.
Is natalizumab overshooting its rebound?
Alteration of CD1 expression in multiple sclerosis.
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