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Most Viewed
[Effect of Chinese Herbs Used in Treating Multiple Sclerosis on T Subsets Using Association Rules].
The Corpus Callosum and Forensic Issues-An Overview.
Is pruritus an indicator of aquaporin-positive neuromyelitis optica?
Immune recognition of citrullinated epitopes.
Multiple sclerosis and vitamin D: an update.
Novel Therapeutic Avenues for Chronic Inflammatory Demyelinating Polyneuropathy: The Difficulties of Disease Diversity.
A multi-time-point modality-agnostic patch-based method for lesion filling in multiple sclerosis.
Pattern and motion-related visual-evoked potentials in neuroborreliosis: follow-up study.
TITLE: VITAMIN D DEFICIENCY IS NOT ASSOCIATED WITH EARLY STAGES OF THYROID AUTOIMMUNITY.
One handed intermittent self-urinary catheterism with an adaptive equipment: Case report of an amputee patient suffering from multiple sclerosis.
Health insurance affects the use of disease-modifying therapy in multiple sclerosis.
Impaired Neurovisceral Integration of Cardiovascular Modulation Contributes to Multiple Sclerosis Morbidities.
Peptide motif for the rat MHC class II molecule RT1.Da: similarities to the multiple sclerosis-associated HLA-DRB1*1501 molecule.
Regulatory T cell function correlates with serum 25-hydroxyvitamin D, but not with 1,25-dihydroxyvitamin D, parathyroid hormone and calcium levels in patients with relapsing remitting multiple sclerosis.
Study of the Peripheral Nerve Fibers Myelin Structure Changes during Activation of Schwann Cell Acetylcholine Receptors.
Resources.
AVN-101: A Multi-Target Drug Candidate for the Treatment of CNS Disorders.
Instructions for Completing Postreading Self-Assessment and CME Test and Tally Sheet.
Characterization of the encephalitogenic immune response in a model of multiple sclerosis.
Vitamin D: not just bone, but also immunity.
Chronic peripheral nerve compression disrupts paranodal axoglial junctions.
The curtain is drawn for both natalizumab and fingolimod (FTY720): a new era of multiple sclerosis therapy has arrived.
I kappa B kinase 2/beta deficiency controls expansion of autoreactive T cells and suppresses experimental autoimmune encephalomyelitis.
Lack of pathogenicity of immunodominant T and B cell determinants of the nicotinic acetylcholine receptor epsilon-chain.
Infiltration of Th1 and Th17 cells and activation of microglia in the CNS during the course of experimental autoimmune encephalomyelitis.
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