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Most Viewed
Options in improving respiratory function in multiple sclerosis.
[Oculomotor system and multiple sclerosis.]
Development of [(3)H]2-Carboxy-4,6-dichloro-1H-indole-3-propionic Acid ([(3)H]PSB-12150): A Useful Tool for Studying GPR17.
PET Imaging of Disease Progression and Treatment Effects in the Experimental Autoimmune Encephalomyelitis Rat Model.
No association between identified multiple sclerosis non-MHC risk loci and neuromyelitis optica.
Bisphenol-A Impairs Myelination Potential During Development in the Hippocampus of the Rat Brain.
Age-dependent differences in microglial responses to systemic inflammation are evident as early as middle age.
Comparison of myelin water fraction values in periventricular white matter lesions between multiple sclerosis and neuromyelitis optica spectrum disorder.
CLIPPERS-like MRI findings in a patient with multiple sclerosis.
Immunity and inflammation in neurodegenerative diseases.
Pediatric demyelinating diseases.
Microglia-mediated neuroinflammation is an amplifier of virus-induced neuropathology.
Interventions for children coping with parental multiple sclerosis: A systematic review.
Fine-tuning of type I IFN-signaling in microglia - implications for homeostasis, CNS autoimmunity and interferonopathies.
Why this is not multiple sclerosis: a case based approach.
Correction: Increased Perfusion in Normal Appearing White Matter in High Inflammatory Multiple Sclerosis Patients.
Role of SIRT1 in autoimmune demyelination and neurodegeneration.
Dynamics and Mechanisms of CNS Myelination.
[New therapeutic options in multiple sclerosis].
Neuroplasticity and motor rehabilitation in multiple sclerosis.
Autonomic Dysregulation in Multiple Sclerosis.
Serum Ferroxidase Activity in Patients with Multiple Sclerosis: A Pilot Study.
Genomic medicine and risk prediction across the disease spectrum.
Corticospinal tract integrity measured using transcranial magnetic stimulation and magnetic resonance imaging in neuromyelitis optica and multiple sclerosis.
Assessment of plasma brain-derived neurotrophic factor (BDNF), activity-dependent neurotrophin protein (ADNP) and vasoactive intestinal peptide (VIP) concentrations in treatment-naïve humans with multiple sclerosis.
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