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Most Viewed
The Disease-Ameliorating Function of Autoregulatory CD8 T Cells Is Mediated by Targeting of Encephalitogenic CD4 T Cells in Experimental Autoimmune Encephalomyelitis.
Biased Treg/Th17 balance away from regulatory toward inflammatory phenotype in relapsed multiple sclerosis and its correlation with severity of symptoms.
Current topics in therapeutic plasmapheresis.
[Low back pain: atypical presentation of multiple sclerosis.]
A profile of childhood neuropathies at a university hospital in Oman.
Interleukin 6 signaling promotes anti-aquaporin 4 autoantibody production from plasmablasts in neuromyelitis optica.
Cognition, cognitive dysfunction, and cognitive rehabilitation in multiple sclerosis.
"I knew what was going to happen if I did nothing and so I was going to do something": Faith, hope, and trust in the decisions of Canadians with multiple sclerosis to seek unproven interventions abroad.
Perforin competent CD8 T cells are sufficient to cause immune-mediated blood-brain barrier disruption.
Neuroplasticity and motor rehabilitation in multiple sclerosis.
Feasibility of Cell Therapy in Multiple Sclerosis: A Systematic Review of 83 Studies.
A subtraction pipeline for automatic detection of new appearing multiple sclerosis lesions in longitudinal studies.
Outcome measures in multiple sclerosis.
[LINGO-1 Expression of brain tissue in Experimental Autoimmune Encephalomyelitis Mouse].
The pathology of multiple sclerosis is the result of focal inflammatory demyelination with axonal damage.
Stride-Time Variability and Fall Risk in Persons with Multiple Sclerosis.
What's in the Literature?
Google Trends: new evidence for seasonality of multiple sclerosis.
Tailoring of therapy for chronic inflammatory demyelinating polyneuropathy.
Distinct roles of T-cell lymphopenia and the microbial flora for gastrointestinal and CNS autoimmunity.
Further evidence for the neuroprotective role of oleanolic acid in a model of focal brain hypoxia in rats.
Myelinating Cultures: An In Vitro Tool to Identify Demyelinating and Axopathic Autoantibodies.
Inflammation negatively regulates FOXP3 and regulatory T-cell function via DBC1.
MHC and non-MHC gene regulation of disease susceptibility and disease course in experimental inflammatory peripheral neuropathy.
Enriched CD161high CCR6+ γδ T Cells in the Cerebrospinal Fluid of Patients With Multiple Sclerosis.
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