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Most Viewed
Predictions for the future of kallikrein-related peptidases in molecular diagnostics.
Matrix Metalloproteinase-8 Plays a Pivotal Role in Neuroinflammation by Modulating TNF-α Activation.
Attention-Deficit/Hyperactivity Disorder in Offspring of Mothers With Inflammatory and Immune System Diseases.
Role of puberty in multiple sclerosis risk and course.
[Pay attention to the differences and similarities in the pathogenesis and management of multiple sclerosis and neuromyelitis optica].
Nuclear export inhibitors avert progression in preclinical models of inflammatory demyelination.
JC virus-iLOV fluorescent strains enable the detection of early and late viral protein expression.
Vitamin D supplementation and antibodies against the Epstein-Barr virus in multiple sclerosis patients.
The role of optical coherence tomography (OCT) in optic neuritis (ON).
Anxiety state affects information processing speed in patients with multiple sclerosis.
Venous endothelial injury in central nervous system diseases.
Signaling through ERK1/2 Controls Myelin Thickness during Myelin Repair in the Adult Central Nervous System.
[Relationship between biochemical parameters of cerebrospinal fluid and the course of multiple sclerosis.]
Clinical Relevance of Brain Volume Measures in Multiple Sclerosis.
Anti-neurofascin antibody in patients with combined central and peripheral demyelination.
Quantitative MRI and ultrastructural examination of the cuprizone mouse model of demyelination.
Transient autoimmune hyperthyroidism following the withdrawal of Natalizumab in patients with multiple sclerosis.
[New and future treatments for neurological disorders--knowledge essential to daily clinics and future prospects. Topics: 8. Guillain-Barré syndrome and chronic inflammatory demyelinating polyneuropathy].
Brain networks disconnection in early multiple sclerosis cognitive deficits: An anatomofunctional study.
In vivo imaging reveals rapid astrocyte depletion and axon damage in a model of neuromyelitis optica-related pathology.
Neurodegeneration: Active astrocytes drive inflammation.
Risk for nevus transformation and melanoma proliferation and invasion during natalizumab treatment: four years of dermoscopic follow-up with immunohistological studies and proliferation and invasion assays.
Targeting of ECM molecules and their metabolizing enzymes and receptors for the treatment of CNS diseases.
Epidemiological characteristics and functional disability of multiple sclerosis patients in kosovo.
Contribution of CD8 T lymphocytes to the immuno-pathogenesis of multiple sclerosis and its animal models.
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