Multiple Sclerosis Discovery Forum

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Benchmarks of meaningful impairment on the MSFC and BICAMS.
Relationship Between Cerebrospinal Fluid Protein Levels and Electrophysiologic Abnormalities in Guillain-Barré Syndrome.
Cortical excitability changes over time in progressive multiple sclerosis.
The major myelin-resident protein PLP is transported to myelin membranes via a transcytotic mechanism: involvement of sulfatide.
Temporal Hierarchical Adaptive Texture CRF for Automatic Detection of Gadolinium-Enhancing Multiple Sclerosis Lesions in Brain MRI.
The GSTP1 gene variant rs1695 is not associated with an increased risk of multiple sclerosis.
Differential Sox10 genomic occupancy in myelinating glia.
The Norwegian Multiple Sclerosis Registry and Biobank.
GM-CSF: An immune modulatory cytokine that can suppress autoimmunity.
Optical coherence tomography and visual evoked potentials: which is more sensitive in multiple sclerosis?
SJL Mice Infected with Acanthamoeba castellanii Develop Central Nervous System Autoimmunity through the Generation of Cross-Reactive T Cells for Myelin Antigens.
Can we switch microglia's phenotype to foster neuroprotection? Focus on multiple sclerosis.
Th22 cells are expanded in multiple sclerosis and are resistant to IFN-β
Progressive resistance therapy is not the best way to rehabilitate deficits due to multiple sclerosis: Yes.
Oral versus intravenous steroids in multiple sclerosis relapses - a perennial question?
The role of TPA I/D and PAI-1 4G/5G polymorphisms in multiple sclerosis.
C-11Utility of the Ten Item Personality Inventory (TIPI) in Multiple Sclerosis (MS): What Can 10 Items Tell Us?
What is the true clinicopathologic spectrum of neuromyelitis optica?-Reply.
Relapse of multiple sclerosis in a patient retaining CCR7-expressing T cells in CSF under fingolimod therapy.
Block of a subset of sodium channels exacerbates experimental autoimmune encephalomyelitis.
Intrathecal baclofen in multiple sclerosis and spinal cord injury: complications and long-term dosage evolution.
Th1-mediated experimental autoimmune encephalomyelitis is CXCR3-independent.
[Interferons and autoimmune diseases].
Treatment strategies for multiple sclerosis: When to start, when to change, when to stop?
Intrathecal CD4(+) and CD8(+) T cell responses to endogenously synthesized candidate disease-associated human autoantigens in multiple sclerosis patients.

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