Multiple Sclerosis Discovery Forum

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Slowing of information processing speed without motor slowing in multiple sclerosis observed during two crossing-off tasks.
Nurr1 reduction influences the onset of chronic EAE in mice.
Extracellular Vesicles in Multiple Sclerosis: What are They Telling Us?
Thalamic atrophy and cognitive impairment in clinically isolated syndrome and multiple sclerosis.
Sexual dysfunction in women with multiple sclerosis: Dimensions and contributory factors.
Correction: Comparative Study on the Therapeutic Potential of Neurally Differentiated Stem Cells in a Mouse Model of Multiple Sclerosis.
RECENT ADVANCES IN MESENCHYMAL STEM CELL IMMUNOMODULATION. THE ROLE OF MICROVESICLES.
General Mathematical Ability Predicts PASAT Performance in MS Patients: Implications for Clinical Interpretation and Cognitive Reserve.
Multiple sclerosis-associated single-nucleotide polymorphisms in CLEC16A correlate with reduced SOCS1 and DEXI expression in the thymus.
Inhibition of delta-like ligand 4 decreases Th1/Th17 response in a mouse model of multiple sclerosis.
[Natalizumab in the treatment of patients with relapsing-remitting multiple sclerosis: the preliminary results of the observational program "Sovet"]
Electrophysiological patterns of oropharyngeal swallowing in multiple sclerosis.
Ascl1/Mash1 Promotes Brain Oligodendrogenesis during Myelination and Remyelination.
Abnormal adaptation over time of motor network recruitment in multiple sclerosis patients with fatigue.
Wavelet-Based Scalar-on-Function Finite Mixture Regression Models.
Effects of albendazole combined with TSII-A (a Chinese herb compound) on optic neuritis caused by Angiostrongylus cantonensis in BALB/c mice.
Downregulation of Sphingosine 1-Phosphate Receptor 1 Promotes the Switch from Tangential to Radial Migration in the OB.
Neurosarcoidosis.
Effects of whole body vibration on muscle spasticity for people with central nervous system disorders: A systematic review.
Current understanding on the role of standard and immunoproteasomes in inflammatory/immunological pathways of multiple sclerosis.
Immunological Mechanism of Action and Clinical Profile of Disease-Modifying Treatments in Multiple Sclerosis.
Corrigendum.
Chemokine receptor expression of T cells in the cerebrospinal fluid of relapsing multiple sclerosis.
Characterizing cognitive function during relapse in multiple sclerosis.
Identification of a microglia phenotype supportive of remyelination.

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