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Most Viewed
Intense inflammation and nerve damage in early multiple sclerosis subsides at older age: a reflection by cerebrospinal fluid biomarkers.
Spring cleaning: time to rethink imaging research lines in MS?
Blockade of CD127 Exerts a Dichotomous Clinical Effect in Marmoset Experimental Autoimmune Encephalomyelitis.
Towards programming immune tolerance through geometric manipulation of phosphatidylserine.
Iron in Multiple Sclerosis and Its Noninvasive Imaging with Quantitative Susceptibility Mapping.
Minocycline protects SH-SY5Y cells from 6-hydroxydopamine by inhibiting both caspase-dependent and -independent programmed cell death.
Meta-analysis of prevalence.
Antibody index test may aid PML diagnosis on natalizumab
Cost-Effectiveness Analysis of OnabotulinumtoxinA (BOTOX(®)) for the Management of Urinary Incontinence in Adults with Neurogenic Detrusor Overactivity: A UK Perspective.
The ICF as a framework to collect and interpret data on the extent and variety of disability in neurological conditions.
The role of immune cells, glia and neurons in white and gray matter pathology in multiple sclerosis.
Effect of radial shock wave therapy on pain and muscle hypertonia: a double-blind study in patients with multiple sclerosis.
Negotiating the radiologically isolated syndrome.
HSV1 and 2 Detection in the CSF of Multiple Sclerosis Patients by Real-time PCR.
Sativex-induced neurobehavioral effects: causal or concausal? A practical advice!
Chromatic visual evoked potentials in young patients with demyelinating disease.
Gender and Uveitis in Patients with Multiple Sclerosis.
Mitochondrial complex I gene variations; as a potential genetic risk factor in pathogenesis of multiple sclerosis.
Glia in the pathogenesis of neurodegenerative diseases.
Brain leukocyte infiltration initiated by peripheral inflammation or experimental autoimmune encephalomyelitis occurs through pathways connected to the CSF-filled compartments of the forebrain and midbrain.
How microglia kill neurons.
Pharmacological inhibition of caspase-2 protects axotomised retinal ganglion cells from apoptosis in adult rats.
How compelling are the data for Epstein-Barr virus being a trigger for systemic lupus and other autoimmune diseases?
Levels of plasma sulfatides C18:0 and C24:1 correlate with disease status in Relapsing-Remitting Multiple Sclerosis.
Axons are injured by antigen-specific CD8(+) T cells through a MHC class I- and granzyme B-dependent mechanism.
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