Multiple Sclerosis Discovery Forum

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Multiple sclerosis and the spinal cord.
[Differential diagnosis of late-stage neuroborreliosis with affection of the central nervous system.]
Multiple sclerosis: TOWER confirms the efficacy of oral teriflunomide in MS.
Fast Whole-Brain Three-dimensional Macromolecular Proton Fraction Mapping in Multiple Sclerosis.
Sex ratio of multiple sclerosis in the National Swedish MS Register (SMSreg).
Remyelination: The True Regeneration of the Central Nervous System.
Neuroendocrine Immunoregulation in Multiple Sclerosis.
Effect of natalizumab on oxidative damage biomarkers in relapsing-remitting multiple sclerosis.
Melanocortins as Innovative Drugs for Ischemic Diseases and Neurodegenerative Disorders: Established Data and Perspectives.
Therapeutic Armamentarium and Health System Coverage of Multiple Sclerosis in Latin America.
Kv1.3 Deletion Biases T Cells toward an Immunoregulatory Phenotype and Renders Mice Resistant to Autoimmune Encephalomyelitis.
Four different synthetic peptides of proteolipid protein induce a distinct antibody response in MP4-induced experimental autoimmune encephalomyelitis.
FDA approval of Isentress (raltegravir)
Atacicept in multiple sclerosis (ATAMS): a randomised, placebo-controlled, double-blind, phase 2 trial.
Sphingolipids in ocular inflammation.
Anatomical Brain Networks on the Prediction of Abnormal Brain States.
Tysabri (natalizumab)
A genetic variant of the anti-apoptotic protein Akt predicts natalizumab-induced lymphocytosis and post-natalizumab multiple sclerosis reactivation.
Placebo power.
Couples coping with multiple sclerosis: a dyadic perspective on the roles of mindfulness and acceptance.
Cross-Immunoreactivity between Bacterial Aquaporin-Z and Human Aquaporin-4: Potential Relevance to Neuromyelitis Optica.
A small-molecule screen yields idiotype-specific blockers of neuromyelitis optica-immunoglobulin G binding to aquaporin-4.
Cannabinoids in the management of difficult to treat pain.
The causal cascade to multiple sclerosis: a model for MS pathogenesis.
HLA-DR15-derived self-peptides are involved in increased autologous T cell proliferation in multiple sclerosis.

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