Multiple Sclerosis Discovery Forum

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The VZV/IE63-specific T cell response prevents herpes zoster in fingolimod-treated patients.
The Brd4 acetyllysine-binding protein is involved in activation of polyomavirus JC.
Fulminant Central Plus Peripheral Nervous System Demyelination without Antibodies to Neurofascin.
A signal-to-noise-ratio-based analysis of multifocal visual-evoked potentials in multiple sclerosis risk assessment.
Glycoengineering of Interferon-β 1a Improves Its Biophysical and Pharmacokinetic Properties.
Analysis of oxidative stress in zebrafish embryos.
In vitro assessment of the direct effect of laquinimod on basic functions of human neural stem cells and oligodendrocyte progenitor cells.
Patients' experiences of adapting to multiple sclerosis: a qualitative study.
Finax: Cheap Buy Cod
Characterizing contrast-enhancing and re-enhancing lesions in multiple sclerosis.
A CB(1)/CB(2) receptor agonist, WIN 55,212-2, exerts its therapeutic effect in a viral autoimmune model of multiple sclerosis by restoring self-tolerance to myelin.
Consensus statements from a panel of u.s. Managed care pharmacists and physicians for management of multiple sclerosis agents.
Immunomodulators for multiple sclerosis may ameliorate spinal bone loss.
Recommendations for the management of multiple sclerosis relapses.
Low-contrast multifocal visual evoked potentials: Identifying more shades of gray in MS.
[Chronic cerebrospinal venous insufficiency--pathophysiology and treament].
Differential diagnosis for cervical spondylotic myelopathy: literature review.
The impact of multiple sclerosis on family members: a review of the literature.
17β-trenbolone, an anabolic-androgenic steroid as well as an environmental hormone, contributes to neurodegeneration.
Severe anemia in a patient with multiple sclerosis treated with natalizumab.
Effect of monthly intravenous cyclophosphamide in rapidly deteriorating multiple sclerosis patients resistant to conventional therapy.
The genetics of multiple sclerosis: an up-to-date review.
Correlation of diffusion and metabolic alterations in different clinical forms of multiple sclerosis.
[Natalizumab and reduction of carbonylated proteins in patients with multiple sclerosis].
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