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Most Viewed Papers
Cellular mechanisms of adaptive myelination: bridging the gap between animal studies and human cognition.
Comparative evaluation of patients' and physicians' satisfaction with interferon beta-1b therapy.
Familial amyotrophic lateral sclerosis with an I104F mutation in the SOD1 gene: Multisystem degeneration with neurofilamentous aggregates and SOD1 inclusions.
Diagnostic Criteria for Psychosomatic Research and Psychosocial Functioning in Multiple Sclerosis.
Zeb2 is essential for Schwann cell differentiation, myelination and nerve repair.
Cutaneous and Mixed Nerve Silent Period Recordings in Symptomatic Paroxysmal Kinesigenic Dyskinesia.
Thalamocortical-auditory network alterations following cuprizone-induced demyelination.
Nerve conduction velocity in CMT1A: what else can we tell?
Increased regional gray matter atrophy and enhanced functional connectivity in male multiple sclerosis patients.
Role of IL-17-producing lymphocytes in severity of multiple sclerosis upon natalizumab treatment.
Recombinant Antibody Fragments For Neurodegenerative Diseases.
[Cell depletion and myoablation for neuroimmunological diseases].
Tiam1/Rac1 complex controls Il17a transcription and autoimmunity.
A human anti-polysialic acid antibody as a potential treatment to improve function in multiple sclerosis patients.
Autoantibodies against glutamate receptor δ2 after allogenic stem cell transplantation.
Antibody-Mediated Autoimmune Encephalitis in Childhood.
Fingolimod-induced leukoencephalopathy in a patient with neuromyelitis optica spectrum disorder.
A Risk Score for Predicting Multiple Sclerosis.
Atf6α deficiency suppresses microglial activation and ameliorates pathology of experimental autoimmune encephalomyelitis.
Efficient presentation of myelin oligodendrocyte glycoprotein peptides but not protein by astrocytes from HLA-DR2 and HLA-DR4 transgenic mice.
Pseudo-Foster Kennedy Syndrome as a Rare Presentation of Vitamin B12 Deficiency.
The role of microRNAs in the pathogenesis of autoimmune diseases.
Glial and neuronal markers in cerebrospinal fluid in different types of multiple sclerosis.
Safety of switching from natalizumab straight into fingolimod in a group of JCV-positive patients with multiple sclerosis.
Schwann cell differentiation inhibits interferon-gamma induction of expression of major histocompatibility complex class II and intercellular adhesion molecule-1.
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