Multiple Sclerosis Discovery Forum

Inspiring Connections

Most Viewed Papers

Commentary on: Predictive value of health-related quality of life in progression of disability and depression in persons with multiple sclerosis: a 3-year study.
Oculomotor Nerve Palsy as a Rare Presentation and First Sign of Multiple Myeloma.
The contribution of neutrophils to CNS autoimmunity.
Conjunctival Lymphoma in a Patient on Fingolimod for Relapsing-Remitting Multiple Sclerosis.
Triad of Risk for Late Onset Alzheimer's: Mitochondrial Haplotype, APOE Genotype and Chromosomal Sex.
Immunomodulatory Effects Mediated by Dopamine.
Cognitive processess and cognitive reserve in multiple sclerosis.
Multiple sclerosis should be treated using a step-down strategy rather than a step-up strategy-YES.
An emerging role for eotaxins in neurodegenerative disease.
Introduction: Do we need multi-stakeholder colloquia in MS?
A study of patients with aggressive multiple sclerosis at disease onset.
The contribution of depressive symptoms to slowness of information processing in relapsing remitting multiple sclerosis.
Author Response.
Common genetic variants in the plasminogen activation pathway are not associated with multiple sclerosis.
Depiction of dentatorubrothalamic tract fibers in patients with Parkinson's disease and multiple sclerosis in deep brain stimulation.
PDCD1 PD-1.3 polymorphism and allergic bronchial asthma in Russian and Buryat patients.
SLE Neuropathy-Anything New?
Neuroscience: A mechanism for myelin injury.
INNOVATIVE THERAPIES REDEFINE TREATMENT GOALS IN MULTIPLE SCLEROSIS.
Rituximab in multiple sclerosis: A retrospective observational study on safety and efficacy.
Myelination.
Deletion of 14-3-3σ sensitizes mice to DMBA/TPA-induced papillomatosis.
BK virus encephalopathy and sclerosing vasculopathy in a patient with hypohidrotic ectodermal dysplasia and immunodeficiency.
Leuprolide Acetate Inhibits Spinal Cord Inflammatory Response in Experimental Autoimmune Encephalomyelitis by Suppressing NF-x03BA;B Activation.
Exploring the cellular basis of human disease through a large-scale mapping of deleterious genes to cell types.

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