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Most Viewed Papers
An unusual presentation of X-linked adrenoleukodystrophy.
Astragaloside IV inhibits microglia activation via glucocorticoid receptor mediated signaling pathway.
Both endoplasmic reticulum and mitochondrial pathways are involved in oligodendrocyte apoptosis induced by capsular hemorrhage.
Cortical Thinning Correlates with Cognitive Change in Multiple Sclerosis but not in Neuromyelitis Optica.
A Preliminary Comparative Assessment of the Role of CD8+ T Cells in Chronic Fatigue Syndrome/Myalgic Encephalomyelitis and Multiple Sclerosis.
Serum peptide reactivities may distinguish neuromyelitis optica subgroups and multiple sclerosis.
Metformin ameliorates the development of experimental autoimmune encephalomyelitis by regulating T helper 17 and regulatory T cells in mice.
Animal modeling an oligodendrogliopathy - multiple system atrophy.
Neurological disorders and celiac disease.
Applying the RE-AIM Framework to Inform the Development of a Multiple Sclerosis Falls-Prevention Intervention.
Hippocampal function is compromised in an animal model of multiple sclerosis.
Pharmacological Alternatives for the Treatment of Neurodegenerative Disorders: Wasp and Bee Venoms and Their Components as New Neuroactive Tools.
NK-DC crosstalk controls the autopathogenic Th17 response through an innate IFN-γ-IL-27 axis.
Teriflunomide: A Once-Daily Oral Medication for the Treatment of Relapsing Forms of Multiple Sclerosis.
Genetic manipulation of microglia during brain development and disease.
[Multiple sclerosis and microRNA].
Axon regeneration impediment: the role of paired immunoglobulin-like receptor B.
An aza-anthrapyrazole negatively regulates Th1 activity and suppresses experimental autoimmune encephalomyelitis.
MAIT cells in autoimmunity, immune mediated diseases and airways disease.
Neurovascular unit dysfunction with blood-brain barrier hyperpermeability contributes to major depressive disorder: a review of clinical and experimental evidence.
Role of phospholipase A(2)s and lipid mediators in secondary damage after spinal cord injury.
Blocking adhesion molecules as therapy for multiple sclerosis: natalizumab.
Detection of varicella-zoster virus DNA during medullary and brainstem relapses in multiple sclerosis.
DNA Subtraction of In Vivo Selected Phage Repertoires for Efficient Peptide Pathology Biomarker Identification in Neuroinflammation Multiple Sclerosis Model.
Elevation of 20-carbon long chain bases due to a mutation in serine palmitoyltransferase small subunit b results in neurodegeneration.
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