Multiple Sclerosis Discovery Forum

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Most Viewed Papers

The determinants of depression in a Romanian cohort of multiple sclerosis patients.
Targeting the 18-kDa translocator protein: recent perspectives for neuroprotection.
Lyme disease associated neuroretinitis - Case report.
Changes of statistical structural fluctuations unveils an early compacted degraded stage of PNS myelin.
Paroxysmal dysarthria-ataxia in remitting-relapsing Bickerstaff's-like encephalitis.
Cerebrospinal fluid analysis in the context of CNS demyelinating diseases.
Anti-myelin antibodies play an important role in the susceptibility to develop PLP-induced EAE.
[Targeted Therapy and Progressive Multifocal Leukoencephalopathy (PML): PML in the Era of Monoclonal Antibody Therapies].
Is serum vitamin D levels associated with disability in patients with newly diagnosed multiple sclerosis?
Acute sensory neuropathy: 2 year follow-up of sensory conduction.
A trifluoromethyl analogue of celecoxib exerts beneficial effects in neuroinflammation.
The cognitive reserve theory in the setting of pediatric-onset multiple sclerosis.
Symptomatic brain involvement as the initial manifestation of neuromyelitis optica.
Cesarean delivery may increase the risk of multiple sclerosis.
Demographic and clinical profile of myelopathy patients.
Myeloid cell distribution and activity in multiple sclerosis.
Validation of the Serbian Version of Multiple Sclerosis Spasticity Scale 88 (MSSS-88).
EDITORIAL Neuroglia as a Central Element of Neurological Diseases: An Underappreciated Target for Therapeutic Intervention.
Neuroinflammation: Modulating mighty microglia.
Acthar gel treatment suppresses acute exacerbations in a murine model of relapsing-remitting multiple sclerosis.
Activation Status of Human Microglia Is Dependent on Lesion Formation Stage and Remyelination in Multiple Sclerosis.
Myeloid-derived suppressor cells in immunity and autoimmunity.
Fingolimod effects on left ventricular function in multiple sclerosis.
Ipilimumab in patients with melanoma and autoimmune disease.
A single-nucleotide polymorphism in serine-threonine kinase 11, the gene encoding liver kinase b1, is a risk factor for multiple sclerosis.

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