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Most Viewed Papers
Glial and neuronal markers in cerebrospinal fluid predict progression in multiple sclerosis.
In vitro myelin formation using embryonic stem cells.
Natalizumab to fingolimod: Questions answered, unanswered, and unasked.
Inflammation negatively regulates FOXP3 and regulatory T-cell function via DBC1.
Pediatric demyelinating diseases.
CD4(+)NKG2D(+) T Cells Exhibit Enhanced Migratory and Encephalitogenic Properties in Neuroinflammation.
Recent evidence for an expanded role of the kynurenine pathway of tryptophan metabolism in neurological diseases.
A case of relapsing-remitting tumour-like inflammation of the central nervous system.
Targeting B cells in the treatment of multiple sclerosis: recent advances and remaining challenges.
Ipilimumab in patients with melanoma and autoimmune disease.
Ultra-high field MTR and qR2* differentiates subpial cortical lesions from normal-appearing gray matter in multiple sclerosis.
Ginsenoside Rb1 attenuates activated microglia-induced neuronal damage.
Current Perspectives on Interferon Beta-1b for the Treatment of Multiple Sclerosis.
Therapies for multiple sclerosis: translational achievements and outstanding needs.
The Outlook for Alemtuzumab in Multiple Sclerosis.
Neurological disorders and celiac disease.
NADH-dehydrogenase Type-2 Suppresses Irreversible Visual Loss and Neurodegeneration in the EAE Animal Model of MS.
MS patients must have access to the best treatment options.
Disutility of Illness for Caregivers and Families: A Systematic Review of the Literature.
[Pulmonary lymphangioleiomyomatosis of unusual revelation during multiple sclerosis].
Regulatory T cell number in multiple sclerosis patients: A meta-analysis.
Tryptophan metabolite analog, N-(3,4-dimethoxycinnamonyl) anthranilic acid, ameliorates acute graft-versus-host disease through regulating T cell proliferation and polarization.
Albumin heterogeneity in low-abundance fluids. The case of urine and cerebro-spinal fluid.
Different Mechanisms Regulate Expression of Zebrafish Myelin P0 in Myelinating Oligodendrocytes and its Induction Following Axonal Injury.
Neuroinflammation: The Devil is in the Details.
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