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Most Viewed Papers
MAIT cells in autoimmunity.
Are There Differences in the Prevalence of Palliative Care-Related Problems in People Living With Advanced Cancer and Eight Non-Cancer Conditions? A Systematic Review.
[Cognitive impairment in multiple sclerosis].
[Regarding the article: "multiple sclerosis treatment with natalizumab: analysis of a hospital-based cohort"].
Flare up reaction during provocation test to glatiramer acetate in a patient with allergy to interferon beta1a.
[A review of multiple sclerosis (1). Presentation of a case.]
Trichuris suis ova in relapsing-remitting multiple sclerosis and clinically isolated syndrome (TRIOMS): study protocol for a randomized controlled trial.
Disease-modifying drugs for multiple sclerosis must be globally available according to therapeutic guidelines suitable to different regions of the world.
Antenarratives to inform health care research: exploring workplace illness disclosure for people with multiple sclerosis (MS).
Location of brain lesions predicts conversion of clinically isolated syndromes to multiple sclerosis.
The role of speed versus working memory in predicting learning new information in multiple sclerosis.
Differentiating patterns of oligoclonal banding in the cerebrospinal fluid and the diagnosis of multiple sclerosis.
Ipsilateral uveitis and optic neuritis in multiple sclerosis.
How do autoimmune diseases cluster in families? A systematic review and meta-analysis.
Adaptive Immune Responses in CNS Autoimmune Disease: Mechanisms and Therapeutic Opportunities.
Safety and efficacy of natalizumab in children with multiple sclerosis.
Is It Too Early to Predict the Failure of Natalizumab in NMO?-Reply.
Neurodegeneration: Active astrocytes drive inflammation.
Delayed nerve stimulation promotes axon-protective neurofilament phosphorylation, accelerates immune cell clearance and enhances remyelination in vivo in focally demyelinated nerves.
Management of fatigue in persons with multiple sclerosis.
Natural Killer Cell Subsets in Cerebrospinal Fluid of Patients with Multiple Sclerosis.
Fibroblast growth factor signalling in multiple sclerosis: inhibition of myelination and induction of pro-inflammatory environment by FGF9.
Nano- and micro-based systems for immunotolerance induction in Multiple Sclerosis.
How strong is the relationship between glaucoma, the retinal nerve fibre layer, and neurodegenerative diseases such as Alzheimer's disease and multiple sclerosis?
Oxidised LDL lipids, statins and a blood-brain barrier.
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