Multiple Sclerosis Discovery Forum

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Most Viewed Papers

Telephone-based physical activity counseling for major depression in people with multiple sclerosis.
Partial MHC class II constructs inhibit MIF/CD74 binding and downstream effects.
Redox processes in neurodegenerative disease involving reactive oxygen species.
POLG mutations associated with remitting/relapsing neurological events.
A Further TWEAK to Multiple Sclerosis Pathophysiology.
Elevated Expression of Fractalkine (CX3CL1) and Fractalkine Receptor (CX3CR1) in the Dorsal Root Ganglia and Spinal Cord in Experimental Autoimmune Encephalomyelitis: Implications in Multiple Sclerosis-Induced Neuropathic Pain.
[Clinical epidemiology of multiple sclerosis in Moscow. Discriptive epidemiology in population of one region of Moscow.]
Dual Targeting of the Chemokine Receptors CXCR4 and ACKR3 with Novel Engineered Chemokines.
Autologous Haematopoietic Stem Cell Transplantation (AHSCT) for Aggressive Multiple Sclerosis - whom, when and how.
Myeloradicular Form of Neuroschistosomiasis in a Six-Year-Old Boy Infected With Schistosoma mansoni.
Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis.
Relationship between Damage to the Cerebellar Peduncles and Clinical Disability in Multiple Sclerosis.
Long-term suppression of EAE relapses by pharmacological impairment of epitope spreading.
Neuroinflammation. Contemporary anti-inflammatory treatment approaches.
Further investigation of safety monitoring guidelines based on magnetic resonance imaging lesion activity in multiple sclerosis clinical trials.
Redefining Self: Patients' Decision Making About Treatment for Multiple Sclerosis.
Axin2 as regulatory and therapeutic target in newborn brain injury and remyelination.
Autologous bone marrow transplantation for the treatment of multiple sclerosis.
Specialty drug coupons lower out-of-pocket costs and may improve adherence at the risk of increasing premiums.
Axonally derived matrilin-2 induces proinflammatory responses that exacerbate autoimmune neuroinflammation.
γδ T cells as early sensors of tissue damage and mediators of secondary neurodegeneration.
Improving communication with multiple sclerosis patients.
Differential Sox10 genomic occupancy in myelinating glia.
Thalamic-hippocampal-prefrontal disruption in relapsing-remitting multiple sclerosis.
IL-17-Producing Vγ4+ γδ T Cells Require Sphingosine 1-Phosphate Receptor 1 for Their Egress from the Lymph Nodes under Homeostatic and Inflammatory Conditions.

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