Multiple Sclerosis Discovery Forum

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Most Viewed Papers

Multiple sclerosis: from molecules to treatment.
How microglia kill neurons.
Genetic inactivation of mitochondria-targeted redox enzyme p66ShcA preserves neuronal viability and mitochondrial integrity in response to oxidative challenges.
Requirement for interleukin-1 to drive brain inflammation reveals tissue-specific mechanisms of innate immunity.
[Kidney function tests in multiple sclerosis].
Multiple sclerosis in Sistan and Baloochestan, South East Iran.
Fibronectin aggregation in multiple sclerosis lesions impairs remyelination.
MANBA, CXCR5, SOX8, RPS6KB1 and ZBTB46 are genetic risk loci for multiple sclerosis.
Patient Characteristics Associated with Medication Adherence.
Anti-idiotypic network induced by T cell vaccination against experimental autoimmune encephalomyelitis.
Affinity Proteomic Profiling of Plasma, Cerebrospinal Fluid, and Brain Tissue within Multiple Sclerosis.
Immunopathological patterns from EAE and Theiler's virus infection: Is multiple sclerosis a homogenous 1-stage or heterogenous 2-stage disease?
IL-12, but not IL-23, induces the expression of IL-7 in microglia and macrophages: Implications for multiple sclerosis.
A selective thyroid hormone β receptor agonist enhances human and rodent oligodendrocyte differentiation.
Metallic gold slows disease progression, reduces cell death and induces astrogliosis while simultaneously increasing stem cell responses in an EAE rat model of multiple sclerosis.
Diurnal fluctuations in brain volume: Statistical analyses of MRI from large populations.
A Discrete Choice Experiment to Determine Patient Preferences for Injection Devices in Multiple Sclerosis.
Syringomyelia coexisting with guillain-barre syndrome.
Glial progenitor cell-based treatment and modeling of neurological disease.
Paradoxical inflammation induced by anti-TNF agents in patients with IBD.
[Disruption of the blood-brain barrier in inflammatory neurological diseases].
Fully automatic detection of deep white matter T1 hypointense lesions in multiple sclerosis.
So many progenitors, so little myelin.
Identification of a unique TGF-β-dependent molecular and functional signature in microglia.
Neuroborreliosis during natalizumab treatment in multiple sclerosis.

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