Multiple Sclerosis Discovery Forum

Inspiring Connections

Most Viewed Papers

Association Between MTHFR Genetic Variants and Multiple Sclerosis in a Southern Iranian Population.
[Current immunotherapy of multiple sclerosis].
Freedom from disease activity in multiple sclerosis.
Altered collagen expression in jugular veins in multiple sclerosis.
Mouse hepatitis virus infection upregulates genes involved in innate immune responses.
Multifocal visual evoked potentials are influenced by variable contrast stimulation in MS.
Self-tolerance in multiple sclerosis.
Hyperconnectivity is a Fundamental Response to Neurological Disruption.
Familial comorbidity of bipolar disorder and multiple sclerosis: Genetic susceptibility, coexistence or causal relationship?
Differential effect of baclofen on cortical and spinal inhibitory circuits.
Natalizumab Therapy for Highly Active Pediatric Multiple Sclerosis.
Targeting NOX enzymes in the central nervous system: therapeutic opportunities.
Neuropathologic correlates of magnetic resonance imaging in multiple sclerosis.
Melatonin Contributes to the Seasonality of Multiple Sclerosis Relapses.
Clinical and pathological effects of intrathecal injection of mesenchymal stem cell-derived neural progenitors in an experimental model of multiple sclerosis.
Serum IL-17F does not predict poor response to IM IFNβ-1a in relapsing-remitting MS.
[Spinal anesthesia for cesarean section in a patient with multiple sclerosis].
Endovascular treatment of CCSVI in patients with multiple sclerosis: clinical outcome of 462 cases.
HERVs in neuropathogenesis.
Symptoms, Prevalence and Impact of Multiple Sclerosis in Younger Patients: A Multinational Survey.
Involvement and interplay of Parkin, PINK1 and DJ1 in neurodegenerative and neuroinflammatory disorders.
Regulation of Neuroinflammation through Programed Death-1/Programed Death Ligand Signaling in Neurological Disorders.
The changing clinical course of multiple sclerosis: A matter of grey matter.
The toxicology of HMG-CoA reductase inhibitors: prediction of human risk.
Amyloid fibrils composed of hexameric peptides attenuate neuroinflammation.

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