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Most Viewed Papers
[Cannabinoid drugs for neurological diseases: what is behind?].
Epistasis and immunity: the role of genetic interactions in autoimmune diseases.
Riluzole blocks persistent Na+ and Ca2+ currents and modulates release of glutamate via presynaptic NMDA receptors on neonatal rat hypoglossal motoneurons in vitro.
A biopsychosocial model of resilience for multiple sclerosis.
Active MS is associated with accelerated retinal ganglion cell/inner plexiform layer thinning.
Oral Agent Outcomes
Changes in Cognitive Processing Speed, Mood, and Fatigue in an Observational Study of Persons With Multiple Sclerosis Treated With Dalfampridine-ER.
New Drug Review 2010: Ustekinumab (Stelara, Centocor Ortho Biotech)
An Empirical Investigation of a Biopsychosocial Model of Pain in Multiple Sclerosis.
The biological significance of brain barrier mechanisms: help or hindrance in drug delivery to the central nervous system?
The natural course of clinically isolated syndrome in pediatric patients.
Association between multiple sclerosis and chronic periodontitis: a population-based pilot study.
Extension Trial Data Shows Cladribine Benefits Persist in RRMS Patients
Huperzine A ameliorates experimental autoimmune encephalomyelitis via the suppression of T cell-mediated neuronal inflammation in mice.
Multiple Sclerosis: Validation of MR Imaging for Quantification and Detection of Iron.
Treatment with laquinimod reduces development of active MRI lesions in relapsing MS.
IFN-γ Signaling to Astrocytes Protects from Autoimmune Mediated Neurological Disability.
Predictors of disease activity in 857 patients with MS treated with interferon beta-1b.
CCSVI and MS: no meaning, no fact.
Acute relapse after initiation of Siponimod in a patient with secondary progressive MS.
IL-1β induces hypomyelination in the periventricular white matter through inhibition of oligodendrocyte progenitor cell maturation via FYN/MEK/ERK signaling pathway in septic neonatal rats.
Seeing optic neuritis through myopic eyes.
Multiple sclerosis: a primary care perspective.
Fatigue and fatigability in neurologic illnesses: Proposal for a unified taxonomy.
Inflammatory Response and Chemokine Expression in the White Matter Corpus Callosum and Gray Matter Cortex Region During Cuprizone-Induced Demyelination.
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