Multiple Sclerosis Discovery Forum

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The value of qualitative and quantitative assessment of lesion to cerebral cortex signal ratio on double inversion recovery sequence in the differentiation of demyelinating plaques from non-specific T2 hyperintensities.
Heterogeneity of multiple sclerosis lesions: implications for the pathogenesis of demyelination.
Avonex approval application
Observations on oligodendrocyte degeneration, the resolution of status spongiosus and remyelination in cuprizone intoxication in mice.
Validation of the Brazilian version of the neurological fatigue index for multiple sclerosis.
Anti-JC virus antibody prevalence in a multinational multiple sclerosis cohort.
Firategrast
UK multiple sclerosis risk-sharing scheme: a new natural history dataset and an improved Markov model.
Adherence to Disease Modifying Drugs among Patients with Multiple Sclerosis in Germany: A Retrospective Cohort Study.
Age-related and cuprizone-induced changes in myelin and transcription factor gene expression and in oligodendrocyte cell densities in the rostral corpus callosum of mice.
Episode 26 with Dr. Tim Kennedy on remyelination and oligodendrocyte development
A study comparing the effectiveness and safety of teriflunomide and interferon beta-1a in patients with relapsing multiple sclerosis (TENERE)
Smoking and increased risk of multiple sclerosis: parallel trends in the sex ratio reinforce the evidence.
Tumor necrosis factor alpha but not interleukin 1 beta mediates neuroprotection in response to acute nitric oxide excitotoxicity.
Cuprizone neurotoxicity in the rat: morphologic observations.
Autoantibodies and pain.
Cerebrospinal fluid markers reveal intrathecal inflammation in progressive multiple sclerosis.
Rituximab does not reset defective early B cell tolerance checkpoints.
Sequential myelin protein expression during remyelination reveals fast and efficient repair after central nervous system demyelination.
Cortical demyelination is prominent in the murine cuprizone model and is strain-dependent.
The Brain as a Target of Inflammatory Processes
Keystone Symposium: Viral Immunity
B1 cells are unaffected by immune modulatory treatment in remitting-relapsing multiple sclerosis patients.
Second International Porto Congress of Multiple Sclerosis
Pathogen-induced human TH17 cells produce IFN-γ or IL-10 and are regulated by IL-1β.

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