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Most Viewed
Disease mechanisms in MS: RNA profiling uncovers two distinct subsets of patients with multiple sclerosis.
Environmental factors and their regulation of immunity in multiple sclerosis.
Biomechanical disorders of foot in multiple sclerosis.
Multiple sclerosis imaging: recent advances.
Hematopoietic progenitors express myelin basic protein and ensheath axons in Shiverer brain.
The T-cell immunoglobulin and mucin domain (Tim) gene family in asthma, allergy, and autoimmunity.
[Diagnosis and treatment of pelvic disorders in patients with multiple sclerosis].
Immunopathological patterns from EAE and Theiler's virus infection: Is multiple sclerosis a homogenous 1-stage or heterogenous 2-stage disease?
On the contribution of Thomas Clifford Allbutt, F.R.S., to the early history of neuromyelitis optica.
The TASK1 channel inhibitor A293 shows efficacy in a mouse model of multiple sclerosis.
A one-year follow-up study of the Symbol Digit Modalities Test (SDMT) and the Paced Auditory Serial Addition Test (PASAT) in relapsing-remitting multiple sclerosis: an appraisal of comparative longitudinal sensitivity.
Blood coagulation protein fibrinogen promotes autoimmunity and demyelination via chemokine release and antigen presentation.
An update on new and emerging therapies for relapsing-remitting multiple sclerosis.
Long-term benefits of induction therapy in NMO: a case report.
The complement cascade: Yin-Yang in neuroinflammation--neuro-protection and -degeneration.
Clinical experience of natalizumab in Crohn's disease patients in a restricted distribution program.
In vivo MR imaging of intercellular adhesion molecule-1 expression in an animal model of multiple sclerosis.
Temporal reorganization to overcome monocular demyelination.
The clinico-radiological paradox in multiple sclerosis: novel implications of lesion size.
[Anti-MOG antibody in different types of immune-mediated optic neuritis].
Myelin repair and functional recovery mediated by neural cell transplantation in a mouse model of multiple sclerosis.
The specific antigen approach in multiple sclerosis: Can it ever be enough?
Stiffness as a presenting symptom of an odd clinical condition caused by multiple sclerosis and myotonia congenita.
Riboflavin Supplementation to Patients with Multiple Sclerosis does not Improve Disability Status nor is Riboflavin Supplementation Correlated to Homocysteine.
Bee Venom Acupuncture Alleviates Experimental Autoimmune Encephalomyelitis by Upregulating Regulatory T Cells and Suppressing Th1 and Th17 Responses.
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