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Most Viewed
Something Old, New, Borrowed, Blue: Anthracenedione Agents for Treatment of Multiple Sclerosis.
XY sex chromosome complement, compared with XX, in the CNS confers greater neurodegeneration during experimental autoimmune encephalomyelitis
Simvastatin interferes with process outgrowth and branching of oligodendrocytes.
Identification of candidate risk gene variations by whole-genome sequence analysis of four rat strains commonly used in inflammation research.
Predictive value of early serum cytokine changes on long-term interferon beta-1a efficacy in multiple sclerosis.
[Uveitis in multiple sclerosis : Overview and perspectives.]
Neuropathic Pain in Multiple Sclerosis Improved With Oral Famciclovir: A Case Report.
Increased Th17 response to myelin peptides in pediatric MS.
Evoked potential tests in clinical diagnosis.
Blocking LINGO-1 as a Therapy to Promote CNS Repair: From Concept to the Clinic.
The multiple sclerosis depression rating scale.
Myelination: Do Astrocytes Play a Role?
Association of smoking with risk of multiple sclerosis: a population-based study.
Disease course and outcome of 15 monocentrically treated natalizumab-associated progressive multifocal leukoencephalopathy patients.
MANBA, CXCR5, SOX8, RPS6KB1 and ZBTB46 are genetic risk loci for multiple sclerosis.
A Multiple Sclerosis-Associated Variant of CBLB Links Genetic Risk with Type I IFN Function.
Treatment of multiple sclerosis in children and its challenges.
Pathology of multiple sclerosis: where do we stand?
Molecular analysis of interleukin-25 exons 1 and 2 and its serum levels in Iranian patients with multiple sclerosis.
Mechanisms and Pharmacology of Neuropathic Pain in Multiple Sclerosis.
A history of multiple sclerosis investigations in Canada between 1850 and 1950.
Accurate determination of blood-brain barrier permeability using dynamic contrast-enhanced T1-weighted MRI: a simulation and in vivo study on healthy subjects and multiple sclerosis patients.
P2Y12 Receptor on the Verge of a Neuroinflammatory Breakdown.
A new type of microglia gene targeting shows TAK1 to be pivotal in CNS autoimmune inflammation.
The meninges: new therapeutic targets for multiple sclerosis.
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