Multiple Sclerosis Discovery Forum

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Systematic localization of common disease-associated variation in regulatory DNA.
[The role of chronic venous insufficiency in the pathogenesis of brain diseases].
Safety and tolerability of cyclophosphamide 'pulses' in multiple sclerosis: a prospective study in a clinical cohort.
Review of the novelties presented at the 29th Congress of the European Committee for Treatment and Research in Multiple Sclerosis (ECTRIMS) (III).
Multiple sclerosis, solitary sclerosis or something else?
Simvastatin inhibits IL-17 secretion by targeting multiple IL-17-regulatory cytokines and by inhibiting the expression of IL-17 transcription factor RORC in CD4+ lymphocytes.
Exploration of the relationships between regional grey matter atrophy and cognition in multiple sclerosis.
Orbital Doppler evaluation of blood flow velocities in optic neuritis.
Clonal Expansion of Mitochondrial DNA Deletions and the Progression of Multiple Sclerosis.
Transgenic inhibition of astroglial NF-kappaB protects from optic nerve damage and retinal ganglion cell loss in experimental optic neuritis.
Cerebral venous sinus thrombosis in the patient with multiple sclerosis associated with congenital antithrombin deficiency.
Multiple sclerosis reactivation postfingolimod cessation: Is it IRIS?
Transcript profiling of different types of multiple sclerosis lesions yields FGF1 as a promoter of remyelination.
[Magnetic reasonance spectroscopy-guided stereotactic biopsy for brain lesion].
Brain atrophy and disability progression in multiple sclerosis patients: a 10-year follow-up study.
Alemtuzumab: The advantages and challenges of a novel therapy in MS.
[Oral disease modifying therapy of multiple sclerosis: the current view.]
Riluzole.
Lipocalin 2 is a novel immune mediator of experimental autoimmune encephalomyelitis pathogenesis and is modulated in multiple sclerosis.
The relationship between upper leg muscle strength and walking capacity in persons with multiple sclerosis.
[Neuromyelitis optica in the elderly: An unusual demyelinating disease.]
Mechanisms in the Development of Multiple Sclerosis Lesions: Reconciling Autoimmune and Neurodegenerative Factors.
No Help from Dalfampridine in MS-Related Optic Neuritis
Upregulation of integrin expression on monocytes in multiple sclerosis patients treated with natalizumab.
Fat-soluble vitamins as disease modulators in multiple sclerosis.

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