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Most Viewed
NMO spectrum presenting as partial myelitis.
Dynamic Development of Glucocorticoid Resistance during Autoimmune Neuroinflammation.
Soluble helminth products suppress clinical signs in murine experimental autoimmune encephalomyelitis and differentially modulate human dendritic cell activation.
Pre-existing T- and B-cell defects in one progressive multifocal leukoencephalopathy patient.
Atypical forms of optic neuritis.
Neurotrophic factors and their effects in the treatment of multiple sclerosis.
Epigenetic changes in neurology: DNA methylation in multiple sclerosis.
Fingolimod treatment in multiple sclerosis leads to increased macular volume.
Treatment of the chronic progressive form of multiple sclerosis with a combination of cyclophosphamide and prednisone.
A Phase 2, Multicenter, Open-label Study of MEDI-551 in Adults With Relapsed or Refractory Chronic Lymphocytic Leukemia (CLL)
Cannabinoids control spasticity and tremor in a multiple sclerosis model.
[Cost of multiple sclerosis in France.]
Feedforward postural control in individuals with multiple sclerosis during load release.
Importance of Human Leukocyte Antigen (HLA) Class I and II Alleles on the Risk of Multiple Sclerosis.
The role of stress-response systems for the pathogenesis and progression of MS.
Vitamin d and multiple sclerosis.
Neuromyelitis optica lesions may inform multiple sclerosis heterogeneity debate.
Natalizumab in spinal multiple sclerosis in a daily clinical setting.
Alemtuzumab Therapy for Multiple Sclerosis.
Safety and tolerability of cyclophosphamide 'pulses' in multiple sclerosis: a prospective study in a clinical cohort.
[The role of chronic venous insufficiency in the pathogenesis of brain diseases].
25-Hydroxycholecalciferol status in plasma is linearly correlated to daily summer pasture time in cattle at 56°N.
Fingolimod (Gilenya) licence extended
[CHANGES IN THE QUALITATIVE AND QUANTITATIVE COMPOSITION OF GUT MICROBIOTA IN RATS DURING EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS].
VIP Deficient Mice Exhibit Resistance to Lipopolysaccharide Induced Endotoxemia with an Intrinsic Defect in Proinflammatory Cellular Responses.
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