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Most Viewed Papers
Natalizumab strongly suppresses cortical pathology in relapsing-remitting multiple sclerosis.
Masitinib (AB1010), a potent and selective tyrosine kinase inhibitor targeting KIT.
Clinically isolated syndrome - Rethinking the diagnosis.
Teriflunomide versus subcutaneous interferon beta-1a in patients with relapsing multiple sclerosis: a randomised, controlled phase 3 trial.
Safety and Clinical Outcomes of Rituximab Treatment in Patients with Multiple Sclerosis and Neuromyelitis Optica: Experience from a National Online Registry (GRAID).
Best practice in the use of natalizumab in multiple sclerosis.
Multiple sclerosis in Hispanics: a study of clinical disease expression.
IL-17 and Th17 Cells.
Oral teriflunomide for patients with relapsing multiple sclerosis (TOWER): a randomised, double-blind, placebo-controlled, phase 3 trial.
Neuroprotection mediated through estrogen receptor-alpha in astrocytes.
Biogen Presents New Anti-LINGO-1 Phase 2 Acute Optic Neuritis Data Demonstrating Neurological Repair
Treatment Choices in Optic Neuritis: Corticosteroids, Intravenous Immunoglobulin, Plasma Exchange, or Other?
Evaluation of No Evidence of Disease Activity in a 7-Year Longitudinal Multiple Sclerosis Cohort.
Management of worsening multiple sclerosis with mitoxantrone: a review.
Experimental models of spontaneous autoimmune disease in the central nervous system.
Cerebrospinal fluid data compilation and knowledge-based interpretation of bacterial, viral, parasitic, oncological, chronic inflammatory and demyelinating diseases. Diagnostic patterns not to be missed in neurology and psychiatry.
The MS Microbiome Consortium (MSMC): an academic multi-disciplinary collaborative effort to elucidate the role of the gut microbiota in MS
[Usefulness of electronic drug registers: Spanish register of patients treated with fingolimod (Gilenya ®)].
Macrophage depletion impairs oligodendrocyte remyelination following lysolecithin-induced demyelination.
Teriflunomide modest help but safe for MS
Demyelination of the hippocampus is prominent in the cuprizone model.
Gene expression in brain during cuprizone-induced demyelination and remyelination.
FGF2 and FGFR1 signaling regulate functional recovery following cuprizone demyelination.
Cost-Effectiveness of Treatments for Relapsing Remitting Multiple Sclerosis: A French Societal Perspective.
Relapse and disability outcomes in patients with multiple sclerosis treated with fingolimod: subgroup analyses of the double-blind, randomised, placebo-controlled FREEDOMS study.
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