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Most Viewed
The search for the target antigens of multiple sclerosis, part 2: CD8+ T cells, B cells, and antibodies in the focus of reverse-translational research.
Comprehensive Immunophenotyping of CSF cells in relapsing-remitting multiple sclerosis patients with daclizumb therapy
Cost-effectiveness analysis of peginterferon beta-1a compared with interferon beta-1a and glatiramer acetate in the treatment of relapsing-remitting multiple sclerosis in the United States.
[Reduction of spastic increased muscle tone in multiple sclerosis by the nonopioid analgesic flupirtine].
Persistency of neutralizing antibodies depends on titer and interferon-beta preparation.
Circulating innate lymphoid cells are unchanged in response to DAC HYP therapy.
Anti-JCV antibody levels in serum or plasma further define risk of natalizumab-associated PML.
Barcelona Pediatric MS and Related Disorders Update Meeting
Evaluation of the 2015 diagnostic criteria for neuromyelitis optica spectrum disorder.
Chemokine-enhanced migration of human peripheral blood mononuclear cells is antagonized by interferon beta-1b through an effect on matrix metalloproteinase-9.
Combination therapy in patients with relapsing-remitting multiple sclerosis (MS) CombiRx
Klotho Is a Neuroprotective and Cognition-Enhancing Protein.
Posturography in MS patients treated with high dose methylprednisolone.
Rituximab (marketed as Rituxan), Information
Comparative pharmacokinetics and pharmacodynamics of two recombinant human interferon beta-1a (IFN beta-1 a) products administered intramuscularly in healthy male and female volunteers.
Multiple sclerosis: Mitoxantrone promotes differential effects on immunocompetent cells in vitro.
Chronic Inflammatory Demyelinating Polyneuropathy Following Anti-TNF-α Therapy With Infliximab for Crohn's Disease.
Spectrum of nontraumatic myelopathies in Ethiopian patients: hospital-based retrospective study.
Neuroimmunology: Clostridium-reactive T cells are implicated in the pathogenesis of neuromyelitis optica.
Voltage-gated potassium channels in multiple sclerosis: Overview and new implications for treatment of central nervous system inflammation and degeneration.
Interferon-beta is a potent promoter of nerve growth factor production by astrocytes.
Cognitive dysfunction in patients with multiple sclerosis treated with different types of interferon beta: A randomized clinical trial.
IFN-beta1a inhibits the secretion of Th17-polarizing cytokines in human dendritic cells via TLR7 up-regulation.
Gpr126 functions in Schwann cells to control differentiation and myelination via G-protein activation.
Autoimmunity in visual loss.
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