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Most Viewed
Recombinant Antibody Fragments For Neurodegenerative Diseases.
Foxo3 Transcription Factor Drives Pathogenic T Helper 1 Differentiation by Inducing the Expression of Eomes.
A Risk Score for Predicting Multiple Sclerosis.
Progressive multifocal leukoencephalopathy after liver transplantation can have favorable or unfavorable outcome.
Erratum to: 'Purkinje cell injury, structural plasticity and fusion in patients with Friedreich's ataxia'.
[Endogenous retroviruses are associated with autoimmune diseases].
Reactive Oxygen Species Derived from NOX3 and NOX5 Drive Differentiation of Human Oligodendrocytes.
Safety and effectiveness assessment of intravenous immunoglobulin in the treatment of relapsing-remitting multiple sclerosis: A meta-analysis.
Cytokine regulation by modulation of the NMDA receptor on astrocytes.
Listeria rhombencephalitis mimicking a demyelinating event in an immunocompetent young patient.
Cognitive performance, fatigue and event-related potentials in patients with clinically isolated syndrome.
Bladder function and falls in individuals with multiple sclerosis.
Chronic inflammatory demyelinating polyneuropathy associated with manic symptoms.
Autologous hematopoietic cell transplantation in multiple sclerosis.
Improvement of neurological disorders in postmenopausal model rats by administration of royal jelly.
Dysregulation of Chemokine/Chemokine Receptor Axes and NK Cell Tissue Localization during Diseases.
The Gas6/TAM System and Multiple Sclerosis.
Increased regional gray matter atrophy and enhanced functional connectivity in male multiple sclerosis patients.
Fatigue and depression in multiple sclerosis: pharmacological and non-pharmacological interventions.
Analyses on the misdiagnoses of 25 patients with unilateral optic nerve sheath meningioma.
Immunotherapy for IgM anti-myelin-associated glycoprotein paraprotein-associated peripheral neuropathies.
Acute disseminated encephalomyelitis following Plasmodium vivax malaria.
Publicly Available Data Provide Evidence against NR1H3 R415Q Causing Multiple Sclerosis.
Quantitative Susceptibility Mapping and R2* Measured Changes during White Matter Lesion Development in Multiple Sclerosis: Myelin Breakdown, Myelin Debris Degradation and Removal, and Iron Accumulation.
Cerebral Microbleeds in Multiple Sclerosis Evaluated on Susceptibility-weighted Images and Quantitative Susceptibility Maps: A Case-Control Study.
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