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[The role of the solar and geomagnetic activity in the etiology of multiple sclerosis: a review of the literature and prospects of research].
Comment on "Thiamine Deficiency Promotes T Cell Infiltration in Experimental Autoimmune Encephalomyelitis: The Involvement of CCL2".
Monoclonal antibody therapy in multiple sclerosis: critical appraisal and new perspectives.
[New therapeutic options in multiple sclerosis].
Two functional promoter polymorphisms of neuregulin 1 gene are associated with progressive forms of multiple sclerosis.
Challenges and opportunities in designing clinical trials for neuromyelitis optica.
The role of the Epstein-Barr virus in the pathogenesis of some autoimmune disorders - Similarities and differences.
Therapeutic Plasma Exchange in Glucocorticosteroid-Unresponsive Patients With Clinically Isolated Syndrome.
The many faces of impaired bladder emptying.
Motor network efficiency and disability in multiple sclerosis.
[Is it always useful to perform lumbar puncture for the diagnosis of multiple sclerosis? Comment].
Deletion of caspase-8 in mouse myeloid cells blocks microglia pro-inflammatory activation and confers protection in MPTP neurodegeneration model.
Endocannabinoids and the Immune System in Health and Disease.
The UK Risk-Sharing Scheme for interferon-beta and glatiramer acetate in multiple sclerosis. Outcome of the year-6 analysis.
[Symptomatic therapy in multiple sclerosis].
Depletion of Olig2 in oligodendrocyte progenitor cells infected by Theiler's murine encephalomyelitis virus.
Intrathecal synthesis in particular types of multiple sclerosis.
Advances in the treatment of relapsing-remitting multiple sclerosis - critical appraisal of fingolimod.
[Additional indicators of the assessment of efficacy of disease modifying drugs (preliminary results).]
Caregiver burden in multiple sclerosis: A structured review of the instruments.
MAPT gene rs1052553 variant is not associated with the risk for multiple sclerosis.
Effects of vaccination with altered Peptide ligand on chronic pain in experimental autoimmune encephalomyelitis, an animal model of multiple sclerosis.
Epstein-Barr virus is a necessary causative agent in the pathogenesis of multiple sclerosis: No.
Multiple sclerosis: Gene-vitamin D interactions likely to influence pathogenesis and risk of relapse in multiple sclerosis.
Brain radiation and possible presentation of multiple sclerosis.
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