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Most Viewed
Employment and Community Living Issues for People with Multiple Sclerosis.
Biotherapeutics for the treatment of multiple sclerosis: hopes and hazards.
Studies in Experimental Autoimmune Encephalomyelitis Do Not Support Developmental Bisphenol A Exposure as an Environmental Factor in Increasing Multiple Sclerosis Risk.
Genetic burden in multiple sclerosis families.
Multiple sclerosis.
Health-related quality of life in multiple sclerosis patients with bladder, bowel and sexual dysfunction.
Identification of Inflammatory Neuronal Injury and Prevention of Neuronal Damage in Multiple Sclerosis: Hope for Novel Therapies?
The plasticity of human Treg and Th17 cells and its role in autoimmunity.
Regulatory dendritic cell therapy: From rodents to clinical application.
Study of fatigue frequency and its association with sociodemographic and clinical variables in patients with multiple sclerosis.
Effects of a virtual reality and treadmill training on gait of subjects with multiple sclerosis: a pilot study.
Longitudinal associations between MRI and cognitive changes in very early MS.
Efficacy and safety of fingolimod (FTY720) in patients with relapsing-remitting multiple sclerosis (FREEDOMS II)
Cellular immune responses in multiple sclerosis patients treated with interferon-beta.
Electroconvulsive therapy in patient with psychotic depression and multiple sclerosis.
STAT4 controls GM-CSF production by both Th1 and Th17 cells during EAE.
Neuroinflammatory TNFα Impairs Memory via Astrocyte Signaling.
Mode of action and clinical studies with fumarates in multiple sclerosis.
Plantarflexor weakness negatively impacts walking in persons with multiple sclerosis more than plantarflexor spasticity.
The role of TPA I/D and PAI-1 4G/5G polymorphisms in multiple sclerosis.
Using perceptive computing in multiple sclerosis - the Short Maximum Speed Walk test.
Glial cells suppress postencephalitic CD8(+) T lymphocytes through PD-L1.
Th22 cells are expanded in multiple sclerosis and are resistant to IFN-β
Th1-mediated experimental autoimmune encephalomyelitis is CXCR3-independent.
Extensive vascular remodeling in the spinal cord of pre-symptomatic experimental autoimmune encephalomyelitis mice; increased vessel expression of fibronectin and the α5β1 integrin.
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