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Most Viewed
RNA-sequencing reveals oligodendrocyte and neuronal transcripts in microglia relevant to central nervous system disease.
Comparison between the cranial magnetic resonance imaging features of neuromyelitis optica spectrum disorder versus multiple sclerosis in Taiwanese patients.
Neuroprotection in Experimental Autoimmune Encephalomyelitis and Progressive Multiple Sclerosis by Cannabis-Based Cannabinoids.
cFLIP is critical for oligodendrocyte protection from inflammation.
Vibsanin B Preferentially Targets HSP90β, Inhibits Interstitial Leukocyte Migration, and Ameliorates Experimental Autoimmune Encephalomyelitis.
Efficacy of lower fingolimod doses in the treatment of MS.
Statins and Neuroprotection: Basic Pharmacology Needed.
Contribution of Functional LILRA3, but Not Nonfunctional LILRA3, to Sex Bias in Susceptibility and Severity of Anti-Citrullinated Protein Antibody-Positive Rheumatoid Arthritis.
Sexual dysfunction in women with multiple sclerosis: Dimensions and contributory factors.
Update on neurodegeneration with brain iron accumulation.
Multiple Sclerosis: Current Knowledge and Future Outlook.
Massive exacerbation of multiple sclerosis after withdrawal and early restart of treatment with natalizumab.
Multiple Sclerosis State of the Art (SMART): A Qualitative and Quantitative Analysis of Therapy's Adherence, Hospital Reliability's Perception, and Services Provided Quality.
[Therapeutic strategy for CIDP (chronic inflammatory demyelinating polyneuropathy)].
Mast cells on the mind: new insights and opportunities.
Gene expression changes underlying cortical pathology: clues to understanding neurological disability in multiple sclerosis.
[Research progresses of monoclonal antibodies therapy in neuromyelitis optica].
Neuregulin-1 controls an endogenous repair mechanism after spinal cord injury.
Characterization of anti-natalizumab antibodies in multiple sclerosis patients.
Cerebrospinal Fluid IL-12p40, CXCL13 and IL-8 as a Combinatorial Biomarker of Active Intrathecal Inflammation.
Increased CD8+ cytotoxic T cell responses to myelin basic protein in multiple sclerosis.
Neurobehavioral disorders.
Resolvin d1, an endogenous lipid mediator for inactivation of inflammation-related signaling pathways in microglial cells, prevents lipopolysaccharide-induced inflammatory responses.
A combination of fluorescent NFAT and H2B sensors uncovers dynamics of T cell activation in real time during CNS autoimmunity.
The use of interferon Beta and glatiramer acetate in multiple sclerosis.
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