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Most Viewed
Role of the Toll Like receptor (TLR) radical cycle in chronic inflammation: possible treatments targeting the TLR4 pathway.
The pathology of multiple sclerosis.
Association of HLA-DRB1*1501 tagging rs3135388 gene polymorphism with multiple sclerosis.
Compressive myelopathy: magnetic resonance imaging findings simulating idiopathic acute transverse myelopathy.
Cyclophosphamide therapy in pediatric multiple sclerosis.
Genetic characterization of gliomas arising in patients with multiple sclerosis.
Disc swelling and mild initial visual acuity loss predict a better short-term visual acuity outcome in bilateral acute optic neuritis.
Low-Voltage-Activated CaV3.1 Calcium Channels Shape T Helper Cell Cytokine Profiles.
Immune reconstitution inflammatory syndrome in a patient treated with natalizumab presenting progressive multifocal leukoencephalopathy.
The validity and reliability of the neurogenic bladder symptom score (NBSS).
Picornavirus infection leading to immunosuppression.
Management of multiple sclerosis during pregnancy and the reproductive years: a systematic review.
Aging, widowhood, multiple sclerosis, and a dog.
Gammaherpesvirus Infection of Human Neuronal Cells.
Warner Chilcott sells Vectrin product line to Medicis
[Case report: a case of chronic inflammatory demyelinating polyneuropathy (CIDP) exacerbated by influenza vaccination].
Combination treatment of Glatiramer Acetate and Minocycline affects phenotype expression of blood monocyte-derived dendritic cells in Multiple Sclerosis patients.
Neuroprotection in Huntington's disease: a 2-year study on minocycline.
Antagonistic activities of the immunomodulator and PP2A-activating drug FTY720 (Fingolimod, Gilenya) in Jak2-driven hematologic malignancies.
Ustekinumab approved for psoriatic arthritis in US, Europe
Whole-Brain Diffusion Tensor Imaging in Correlation to Visual-Evoked Potentials in Multiple Sclerosis: A Tract-Based Spatial Statistics Analysis.
Abrogation of β-Catenin Signaling in Oligodendrocyte Precursor Cells Reduces Glial Scarring and Promotes Axon Regeneration after CNS Injury.
Neuroaxonal regeneration is more pronounced in early multiple sclerosis than in traumatic brain injury lesions.
Immune-related GTPase Irgm1 exacerbates experimental auto-immune encephalomyelitis by promoting the disruption of blood-brain barrier and blood-cerebrospinal fluid barrier.
Estrogen receptor-β ligand treatment after disease onset is neuroprotective in the multiple sclerosis model.
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