Multiple Sclerosis Discovery Forum

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Monocytes and CD4+ T cells contribution to the under-expression of NR4A2 and TNFAIP3 genes in patients with multiple sclerosis.
Complement activation in multiple sclerosis plaques: an immunohistochemical analysis.
Epidural analgesia and cesarean delivery in multiple sclerosis post-partum relapses: the Italian cohort study.
Suppression of Experimental Autoimmune Optic Neuritis by the Novel Agent Fingolimod.
Central Nervous System and its Disease Models on a Chip.
Evaluation of the immunosuppressive effects of cyclophosphamide in patients with multiple sclerosis.
Optic Neuritis Probably Induced by Isotretinoin.
White matter volume is decreased in the brain of patients with neuromyelitis optica.
Posterior brain damage and cognitive impairment in pediatric multiple sclerosis.
Update on the Autoimmune Pathology of Multiple Sclerosis: B-Cells as Disease-Drivers and Therapeutic Targets.
A genome-wide association study in progressive multiple sclerosis.
Urinary JCV-DNA Testing during Natalizumab Treatment May Increase Accuracy of PML Risk Stratification.
Harnessing monocyte-derived macrophages to control central nervous system pathologies: No longer 'if' but 'how'
Epstein-Barr virus, latitude and multiple sclerosis.
Contributions of vitamin D response elements and HLA promoters to multiple sclerosis risk.
Retinol levels are associated with magnetic resonance imaging outcomes in multiple sclerosis.
IL-1 signal affects both protection and pathogenesis of virus-induced chronic CNS demyelinating disease.
UCSF Neuroimmunology Research Seminar
ATX-MS-1467 in Patients With Relapsing Forms of Multiple Sclerosis
Yoga for multiple sclerosis: a systematic review and meta-analysis.
Center for Drug Evaluation and Research Approval Letter, application number: 21-897
Riluzole inhibits spontaneous Ca2+ signaling in neuroendocrine cells by activation of K+ channels and inhibition of Na+ channels.
Progressive multifocal leukoencephalopathy: new concepts.
Insufficient OPC migration into demyelinated lesions is a cause of poor remyelination in MS and mouse models.
Preventive healthcare use among males with multiple sclerosis.

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