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Most Viewed Papers
T Helper Cells and Cytokine Networks in the Immunopathogenesis of Multiple Sclerosis.
A combined therapeutic approach: extracorporeal shock wave therapy and botulinum toxin in multiple sclerosis-related spasticity.
Single-cell RNA profiling of oligodendroglial lineage cells derived from iPSCs carrying Parkinson's disease-relevant LRRK2-G2019S mutation.
Preventing physical and chemical degradation of the LABL-Fc-MOG, a bifunctional peptide inhibitor, with formulation development approaches.
Philippine Clinical Practice Guidelines for Periodic Health Examination: Lifestyle Interventions for Adults and Adolescents.
HLA-DRB1*15:01 drives sex- and age-dependent microglial immune phenotypes and neuroimmune signaling.
Autoimmune GFAP astrocytopathy with eosinophils on cerebrospinal fluid cytology and isolated spinal cord lesions on MRI: a case report.
Multiple sclerosis patients under treatment with interferon β1-a or ocrelizumab exhibit different T and B cell responses to SARS-CoV-2 vaccine.
Marked Neutrophilic Cerebrospinal Fluid Pleocytosis Following Intravenous Immunoglobulin Mimicking Bacterial Meningitis.
Atypical Presentation of Rapidly Progressive Guillain-Barré Syndrome With Dysautonomia in an Adolescent With Persistent Infection: A Case Report.
Inter-centre heterogeneity, temporal evolution, and factors associated with treatment selection and outcomes in chronic inflammatory demyelinating polyradiculoneuropathy: a multicentre, combined prospective and retrospective observational study.
COVID-19 Vaccine-triggered Relapsing Immune Dysregulation: An Observational Study with Four-year Follow-up.
Spinal cord transverse section hinders Wallerian degeneration and neoangiogenesis after peripheral nerve transection: Involvement of Schwann cells' miR-134-5p.
Graded hypergravity is associated with large-scale reorganization of resting-state cortical networks: EEG evidence from a neurological inactivity phenotype.
Retinal hyper-reflective foci link retinal and cortical pathology in paediatric multiple sclerosis.
CTSS regulates macrophage lipid metabolic reprogramming and white matter repair after intracerebral hemorrhage.
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