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Most Viewed Papers
Update on reproductive safety of current and emerging disease-modifying therapies for multiple sclerosis.
Iran mass produces MS drug Ziferon
Timing of retinal neuronal and axonal loss in MS: a longitudinal OCT study.
Statement on a nonproprietary name adopted by the USAN Council
Testicular hypofunction and multiple sclerosis risk: a record-linkage study.
Impaired NK-mediated regulation of T-cell activity in multiple sclerosis is reconstituted by IL-2 receptor modulation.
Contribution of the oligodendrocyte lineage to CNS repair and neurodegenerative pathologies.
Interferon-beta(1b) treatment in neuromyelitis optica.
The Role of Endogenous IFN-β in the Regulation of Th17 Responses in Patients with Relapsing-Remitting Multiple Sclerosis.
Prediction of oxygen uptake during walking in ambulatory persons with multiple sclerosis.
Limiting multiple sclerosis related axonopathy by blocking Nogo receptor and CRMP-2 phosphorylation.
Subclinical myocardial dysfunction in multiple sclerosis patients remotely treated with mitoxantrone: evidence of persistent diastolic dysfunction.
Pharmacokinetics and pharmacodynamics of the interferon-betas, glatiramer acetate, and mitoxantrone in multiple sclerosis.
Identical lesion morphology in primary progressive and relapsing-remitting MS -an ultrahigh field MRI study.
Antibodies to MOG are transient in childhood acute disseminated encephalomyelitis.
Individual oligodendrocytes have only a few hours in which to generate new myelin sheaths in vivo.
FDA Drug Safety Communication: FDA warns about case of rare brain infection PML with MS drug Tecfidera (dimethyl fumarate)
Divergent Trends of Anti-JCPyV Serum Reactivity and Neutralizing Activity in Multiple Sclerosis (MS) Patients during Treatment with Natalizumab.
Mitoxantrone repression of astrocyte activation: Relevance to multiple sclerosis.
Guillain-Barré Syndrome (42 Cases) Occurring During a Zika Virus Outbreak in French Polynesia.
Transdermal Application of Myelin Peptides in Multiple Sclerosis Treatment.
Beginning of the end of two-stage theory purporting that inflammation then degeneration explains pathogenesis of progressive multiple sclerosis.
Modulation of immune function occurs within hours of therapy initiation for multiple sclerosis.
Attenuation of 7-ketocholesterol-induced overproduction of reactive oxygen species, apoptosis, and autophagy by dimethyl fumarate on 158N murine oligodendrocytes.
Rapid exacerbation of neuromyelitis optica after rituximab treatment.
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