Multiple Sclerosis Discovery Forum

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Most Viewed Papers

Cerebrospinal fluid lactate is associated with multiple sclerosis disease progression.
Investigation of Sequential Growth Factor Delivery during Cuprizone Challenge in Mice Aimed to Enhance Oligodendrogliogenesis and Myelin Repair.
The Impact of Dynamic Balance Measures on Walking Performance in Multiple Sclerosis.
Pediatric demyelinating diseases.
T cell-depleted splenocytes from mice pre-immunized with neuroantigen in incomplete Freund's adjuvant involved in protection from experimental autoimmune encephalomyelitis.
Acute disseminated encephalomyelitis: current knowledge and open questions.
Cryoglobulinaemia (IgG-κ-type and IgM-γ-type) with Occluding Leukocytoclastic Vasculitis in a Patient with Vitiligo and Demyelinating Polyneuropathy.
Kinetics of IL-6 production defines T effector cell responsiveness to regulatory T cells in multiple sclerosis.
Anatomical and functional retinal changes in multiple sclerosis.
Towards an endophenotype in multiple sclerosis.
Relapses in multiple sclerosis: effects of high-dose steroids on cortical excitability.
Cervical and ocular vestibular evoked myogenic potentials in multiple sclerosis participants.
Chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) associated to hereditary neuropathy with liability to pressure palsies (HNPP) and revealed after influenza AH1N1 vaccination.
A Systematic Review of Patients' Perspectives on the Subcutaneous Route of Medication Administration.
Synergistic Interactions between Cytokines and AVP at the Blood-CSF Barrier Result in Increased Chemokine Production and Augmented Influx of Leukocytes after Brain Injury.
MicroRNA let-7e is associated with the pathogenesis of experimental autoimmune encephalomyelitis.
Neuroinflammation and demyelination from the point of nitrosative stress as a new target for neuroprotection.
Bi-Allelic TCRα or β Recombination Enhances T Cell Development but Is Dispensable for Antigen Responses and Experimental Autoimmune Encephalomyelitis.
Neuroinflammation: The Devil is in the Details.
Is total duration of distal compound muscle action potential better than negative peak duration in the diagnosis of chronic inflammatory demyelinating polyneuropathy?
Recessively transmitted predominantly motor neuropathies.
Abrogation of T cell quiescence characterizes patients at high risk for multiple sclerosis after the initial neurological event.
Ipilimumab in patients with melanoma and autoimmune disease.
[Assessment of the size of the thalamus as a method for the evaluation of the activity of neurodegenerative process after the treatment with cerebrolisin in young patients with multiple sclerosis.]
Subretinal angiostrongyliasis-induced optic neuritis.

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