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Most Viewed Papers
High-mobility group box 1 up-regulates aquaporin 4 expression via microglia-astrocyte interaction.
Glial influence on the blood brain barrier.
Multiple sclerosis presenting as a relapsing amnestic syndrome.
Differences in oligoclonal bands and visual evoked potentials in patients with radiologically and clinically isolated syndrome.
Distinction between MOG antibody-positive and AQP4 antibody-positive NMO spectrum disorders.
Fluoxetine prevents oligodendrocyte cell death by inhibiting microglia activation after spinal cord injury.
Neuroborreliosis during natalizumab treatment in multiple sclerosis.
The fatigue-motor performance paradox in multiple sclerosis.
Voltage-gated potassium channel antibodies: Game over.
Perception of affective prosody in patients at an early stage of relapsing-remitting multiple sclerosis.
Clarification on neuromuscular conditions in careers article.
Electrodiagnostic criteria for Guillain-Barrè syndrome: A critical revision and the need for an update.
Aquaporin-4 Autoantibodies in Neuromyelitis Optica: AQP4 Isoform-Dependent Sensitivity and Specificity.
Neuroimmunological diseases and pregnancy.
Hematopoietic progenitors express myelin basic protein and ensheath axons in Shiverer brain.
Short-term evolution of spinal cord damage in multiple sclerosis: a diffusion tensor MRI study.
Vestibular-evoked myogenic potentials, clinical evaluation, and imaging findings in multiple sclerosis.
The onset location of neuromyelitis optica spectrum disorder predicts the location of subsequent relapses.
Acute demyelinating encephalomyelitis in a child following malaria.
Ceramide galactosyltransferase expression is regulated positively by Nkx2.2 and negatively by OLIG2.
Association Between Physical Fitness and Cognitive Function in Multiple Sclerosis: Does Disability Status Matter?
Clostridium perfringens Epsilon Toxin Causes Selective Death of Mature Oligodendrocytes and Central Nervous System Demyelination.
The diadenosine homodinucleotide P18 improves in vitro myelination in experimental Charcot-Marie-Tooth type 1A.
Imipramine and fluoxetine inhibit LPS-induced activation and affect morphology of microglial cells in the rat glial culture.
Therapy with natalizumab is associated with high JCV seroconversion and rising JCV index values.
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