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Most Viewed Papers
Our history
Endogeneous Remyelination: Findings in Human Studies.
Response to disease modifying therapies in African Americans with multiple sclerosis.
Pregnancy and Multiple Sclerosis (MS): A Beneficial Association. Possible therapeutic application of embryo-specific Pre-implantation Factor (PIF*).
Axonal damage in acute multiple sclerosis lesions.
Mast cells: an expanding pathophysiological role from allergy to other disorders.
HyperCKemia Related to the Initial and Recurrent Attacks of Neuromyelitis Optica.
Please mind the gap: axonal transport deficits in multiple sclerosis neurodegeneration.
Cumulative review of thrombotic microangiopathy, thrombotic thrombocytopenic purpura and hemolytic uremic syndrome reports with SC interferon beta-1a.
Plasma chemerin levels in patients with multiple sclerosis.
Strategies for myelin regeneration: lessons learned from development.
Fatigue in Multiple Sclerosis: Neural Correlates and the Role of Non-Invasive Brain Stimulation.
The role of tumour necrosis factor α in the pathogenesis of multiple sclerosis, and pentoxifylline as a possible pharmacotherapeutic intervention.
Urinary JCV-DNA Testing during Natalizumab Treatment May Increase Accuracy of PML Risk Stratification.
Remote effects of intermittent theta burst stimulation of the human pharyngeal motor system.
Conditional DC depletion does not affect priming of encephalitogenic Th cells in EAE.
The role of complementary and alternative medicine in therapy of multiple sclerosis.
Influence of treatments in multiple sclerosis disability: A cohort study.
Significance of Virchow-Robin Spaces in Patients Newly Diagnosed with Multiple Sclerosis: A Case-Control Study in an Arab Population.
Smoking induces long-lasting effects through a monoamine-oxidase epigenetic regulation.
Wnt Signaling in the Pathogenesis of Multiple Sclerosis-Associated Chronic Pain.
Added benefit of fampridine is not proven
No MRI evidence of cortical lesions in neuromyelitis optica.
[2012: Update on diagnosis and treatment of multiple sclerosis].
Default network activity is a sensitive and specific biomarker of memory in multiple sclerosis.
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